Premature ventricular contraction (PVC), also known as ventricular premature beat (VPB) or extrasystole, is a form of irregular heartbeat in which the ventricle contracts prematurely. This results in a "skipped beat" followed by a stronger beat. Individuals with the condition may report feeling that his or her heart "stops" after an attack. PVCs are also called heart palpitations (although there are many other forms of arrhythmia). The depolarization begins in the ventricle instead of the usual place, i.e. the sinus node. PVCs can be a useful natural probe, since they induce Heart rate turbulence whose characteristics can be measured, and used to evaluate cardiac function.
In children, PVCs occur less frequently than in adults, although healthy children are known to have episodes of PVC. In fact, on routine monitoring of children aged 10-13 years with a Holter monitor, about 20% of healthy boys had occurrences of PVC. In otherwise healthy newborns, PVCs will often times resolve on their own by the 12th week of life, and almost never require treatment.
Heart conditions or a previous history of heart attack, ischemia, myocarditis, dilated or hypertrophic cardiomyopathy, myocardial contusion, atrial fibrillation and mitral valve prolapse may cause PVC. Patients with hypomagnesemia, hypokalemia, and hypercalcemia may also present with PVC.
PVCs in young children are thought to be associated with developmental factors of the autonomic nervous system. In older children, sympathomimetic drugs, such as cold or asthma medication may cause PVCs, along with mild cases of viral myocarditis, and excessive caffeine.
In healthy individuals, PVCs can often be resolved with continuous rehydration and by reinstating the balance of magnesium, calcium and potassium within the body.
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